Independent public reference library

Ageing biology, biomarkers, interventions, and research literacy.

Hayflick Limit

Definition

The Hayflick limit is the observation that many normal human cells can divide only a limited number of times in culture before entering a state of permanent growth arrest. This finding challenged the older assumption that normal cultured cells could divide indefinitely and helped establish replicative senescence as a biological phenomenon. [1] [2] [3]

Why It Matters in Ageing Research

The Hayflick limit matters because it links cell division history, telomere shortening, cellular senescence, and tissue renewal. As many somatic cells divide, telomeres can become progressively shorter, eventually contributing to DNA-damage signalling and stable cell-cycle arrest. This makes the Hayflick limit a foundational idea in cellular ageing, although organismal ageing is much broader than the division limit of cells grown in a dish. [4] [5] [6]

Common Confusion

Related Reading

References

  1. Hayflick, L., & Moorhead, P. S. (1961). The serial cultivation of human diploid cell strains. https://doi.org/10.1016/0014-4827(61)90192-6
  2. Hayflick, L. (1965). The Limited in Vitro Lifetime of Human Diploid Cell Strains. https://doi.org/10.1016/0014-4827(65)90211-9
  3. Campisi, J., & d'Adda di Fagagna, F. (2007). Cellular senescence: when bad things happen to good cells. https://doi.org/10.1038/nrm2233
  4. Harley, C. B., Futcher, A. B., & Greider, C. W. (1990). Telomeres shorten during ageing of human fibroblasts. https://doi.org/10.1038/345458a0
  5. Bodnar, A. G., et al. (1998). Extension of life-span by introduction of telomerase into normal human cells. https://doi.org/10.1126/science.279.5349.349
  6. Lopez-Otin, C., et al. (2013). The Hallmarks of Aging. https://doi.org/10.1016/j.cell.2013.05.039
Note

This glossary entry is provided for educational purposes only and does not constitute medical advice.